Se-methylselenocysteine Inhibits Migration and Glycolysis in Anaplastic Thyroid Carcinoma Cells via the ERK1/2 Signaling Pathway <i>in vitro</i>.

These findings indicated that MSC inhibited the viability, migration, and glycolysis of ATC cells via the ERK1/2 signaling pathway, suggesting that MSC may represent a novel therapeutic agent for ATC.

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PubMed
Published
2024-11-01
Primary Topic
Selenium in Biological Systems
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article
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article

Se-methylselenocysteine Inhibits Migration and Glycolysis in Anaplastic Thyroid Carcinoma Cells via the ERK1/2 Signaling Pathway in vitro.

Bixiu Ban, Yuping Liu, Zuojie Luo, Haiyan Yang
PubMed
Selenium in Biological Systems
article

Se-methylselenocysteine Inhibits Migration and Glycolysis in Anaplastic Thyroid Carcinoma Cells via the ERK1/2 Signaling Pathway in vitro.

Bixiu Ban, Yuping Liu, Zuojie Luo, Haiyan Yang
article en

Abstract

These findings indicated that MSC inhibited the viability, migration, and glycolysis of ATC cells via the ERK1/2 signaling pathway, suggesting that MSC may represent a novel therapeutic agent for ATC.

PubMedVol. 54(6)
Guangxi Medical University (CN), First Affiliated Hospital of GuangXi Medical University (CN)
Openalex Percentile: Top 69%
Selenium in Biological Systems
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Se-methylselenocysteine Inhibits Migration and Glycolysis in Anaplastic Thyroid Carcinoma Cells via the ERK1/2 Signaling Pathway <i>in vitro</i>. — Bixiu Ban, Yuping Liu, et al. · PubMed (2024) | TGRS Research Map | TGRS