Clinical case of an atypical course of systemic vasculitis

Vasculitis is a group of diseases based on immunopathological inflammation of blood vessels (arteries, arterioles, capillaries, venules, and veins). As a rule, the consequence of this disease is a change in the structure and function of the organs supplied with blood by the inflamed vessels. The causes of the disease remain unknown. A combination of genetic predisposition with environmental factors, as well as infections involving Staphylococcus aureus or hepatitis virus, are suspected. Vasculitis is fundamentally divided into primary, caused by inflammation of the vessels themselves, and secondary, in which inflammation of the vessel is a reaction to another disease. The main clinical forms of systemic vasculitis are hemorrhagic vasculitis (Henoch-Schonlein purpura), microscopic polyangiitis, granulomatosis with polyangiitis (Wegener’s granulomatosis), eosinophilic granulomatosis with polyangiitis (Churg-Strauss syndrome), polyarteritis nodosa, Kawasaki disease, Takayasu arteritis, etc.