Abstract 320: Length of Transverse Sinus Covered by Stent is an Independent Predictor of Improved Visual Outcomes Following Venous Sinus Stenting in Patients with Idiopathic Intracranial Hypertension

Acute causes of increased intracranial pressures are not known to cause the characteristic venous stenosis or symptomatology seen in idiopathic intracranial hypertension (IIH), posing the question, why elevated CSF pressures in IIH cause venous stenosis. Chronically elevated CSF pressures in IIH may potentially cause thinning and weakening of the dural walls covering these sinuses. This can increase their compliance and susceptibility to compression by even submaximally elevated CSF pressures. Following transverse‐sigmoid junction (TSJ) stenting, the weak, unstented, middle and medial/distal segments of the TS remains vulnerable to future compression. Consequently, we more commonly see distal stent‐adjacent restenosis. This could explain suboptimal improvement, persistent or relapsing IIH symptomatology. Increasing the length of TS stented may lead to comprehensive and effective relief from IIH symptoms. We investigate if 1) higher percentage length of TS stented(%TSS) and 2) distance between distal stent and torcula(TorcDS) significantly predict headache, visual and papilledema resolution after TSJ stenting.