FOXL2 Knockdown Inhibits the Progression of Endometriosis
FOXL2 participates in the occurrence and development of EM through promoting epithelial-mesenchymal transition procession and enhancing the migration and invasion of EC-ESCs, suggesting that FOXL2 may be a new therapeutic target for the EM therapy.
Authors
- Shanshan Cong (ORCID: https://orcid.org/0000-0002-4935-6775)
- Shujie Zhao (ORCID: https://orcid.org/0000-0002-2820-2232)
- Hua Yuan (ORCID: https://orcid.org/0000-0002-5855-2196)
- Xiao‐Jun Yang (ORCID: https://orcid.org/0000-0001-5143-3357)
- Shuang Li (ORCID: https://orcid.org/0000-0001-7414-630X)
- Bing Zhang (ORCID: https://orcid.org/0000-0002-1741-0968)
Institutions
- Jiangnan University (CN)
- Soochow University (CN)
- First Affiliated Hospital of Soochow University (CN)
Publication Details
- Journal
- American Journal of Reproductive Immunology
- Published
- 2025-01-01
- DOI
- https://doi.org/10.1111/aji.70043
- Citations
- 3
- Primary Topic
- Endometriosis Research and Treatment
- Type
- article
- Field-Weighted Citation Impact
- 5.09
Funders
- Jiangnan University