Bindarit attenuates neuroinflammation after subarachnoid hemorrhage by regulating the CCL2/CCR2/NF-κB pathway
Endogenous CCL2 expression was upregulated in the rat SAH model. Bindarit improved neurological functions and reduced neuroinflammation by regulating the CCL2/CCR2/NF-κB pathway in early brain injury after SAH.
Authors
- Pei Wu (ORCID: https://orcid.org/0000-0003-0967-571X)
- Huaizhang Shi (ORCID: https://orcid.org/0000-0003-3150-6234)
- Qiaowei Wu (ORCID: https://orcid.org/0000-0001-9454-1411)
- Kaikun Yuan
- Y. Liu (ORCID: https://orcid.org/0009-0004-1340-0545)
- Yanting Yao
- Anyu Xu (ORCID: https://orcid.org/0009-0003-4366-1132)
- Wei Xu (ORCID: https://orcid.org/0000-0003-0023-4045)
- Hongli Yu (ORCID: https://orcid.org/0000-0002-8487-5079)
- Chao Xu (ORCID: https://orcid.org/0000-0003-4290-3286)
- Zhiyong Ji (ORCID: https://orcid.org/0000-0002-5462-5298)
Institutions
- Jiamusi University (CN)
- Harbin Medical University (CN)
- First Affiliated Hospital of Harbin Medical University (CN)
Publication Details
- Journal
- Brain Research Bulletin
- Published
- 2024-12-31
- DOI
- https://doi.org/10.1016/j.brainresbull.2024.111183
- Citations
- 6
- Primary Topic
- Neuroinflammation and Neurodegeneration Mechanisms
- Type
- article
- Field-Weighted Citation Impact
- 1.48
Funders
- Key Project of Research and Development Plan of Hunan Province
- Key Research and Development Program of Heilongjiang
- National Natural Science Foundation of China