[High expression of AURKB promotes malignant phenotype of osteosarcoma cells by activating nuclear factor-κB signaling <i>via</i> DHX9].

AURKB overexpression promotes the malignant phenotype of osteosarcoma cells by activating the NF-κB signaling pathway via regulating DHX9.

Authors

Institutions

Publication Details

Journal
PubMed
Published
2024-12-20
DOI
https://doi.org/10.12122/j.issn.1673-4254.2024.12.06
Primary Topic
NF-κB Signaling Pathways
Type
article
Controls
|||
ALL TIME
JAN
FEB
MAR
APR
MAY
JUN
JUL
AUG
SEP
OCT
article

[High expression of AURKB promotes malignant phenotype of osteosarcoma cells by activating nuclear factor-κB signaling via DHX9].

Weilai Tong, Feng Yang, Jiangbo Nie, Xinsheng Xie et al.
PubMed
NF-κB Signaling Pathways
article

[High expression of AURKB promotes malignant phenotype of osteosarcoma cells by activating nuclear factor-κB signaling via DHX9].

Weilai Tong, Feng Yang, Jiangbo Nie, Xinsheng Xie, Yanxin Zhong, Yu Liu, Jiaming Liu, Zhili Liu
article en

Abstract

AURKB overexpression promotes the malignant phenotype of osteosarcoma cells by activating the NF-κB signaling pathway via regulating DHX9.

PubMedVol. 44(12)
Nanchang University (CN), Jiangxi Provincial People's Hospital (CN), Second Hospital of Nanchang (CN), First Affiliated Hospital of Nanchang University (CN)
NF-κB Signaling Pathways
AI Navigator

Ask Laika to Summarize, Analyze, and Connect papers live on the map.

Summarize Papers & Methodologies

Extract key findings, datasets, and comparative methods across publications.

Benchmark Rankings & Visual Analytics

Rank top research institutions, authors, funders, topics, and journals by Field-Weighted Citation Impact (FWCI) and paper volume with instant charts.

Connect Distant Disciplines

Bridge topological clusters on the map to find hidden collaborative intersections.