Regulation and Function of CCL2 and N-Myc in Retinoic Acid-treated Neuroblastoma Cells

CCL2 and N-Myc promote the viability of RA-treated cells, although the levels of these mediators were not consistently correlated with cellular outcomes, especially during apoptotic signaling.

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Publication Details

Journal
Cancer Genomics & Proteomics
Published
2024-12-27
DOI
https://doi.org/10.21873/cgp.20490
Citations
1
Primary Topic
Neuroblastoma Research and Treatments
Type
article
Field-Weighted Citation Impact
0.37
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article

Regulation and Function of CCL2 and N-Myc in Retinoic Acid-treated Neuroblastoma Cells

Vicki Waetzig, Ingolf Cascorbi, BERIT SCHMIDT, Meike Kaehler et al.
1 citations
Cancer Genomics & Proteomics
Neuroblastoma Research and Treatments
0.37
article

Regulation and Function of CCL2 and N-Myc in Retinoic Acid-treated Neuroblastoma Cells

Vicki Waetzig, Ingolf Cascorbi, BERIT SCHMIDT, Meike Kaehler, NINA SOPHIE POMMERT, Henrike Bruckmueller, Reema Sami Issa, NANKE MURRA, VERA TIM
article en
1 citations

Abstract

CCL2 and N-Myc promote the viability of RA-treated cells, although the levels of these mediators were not consistently correlated with cellular outcomes, especially during apoptotic signaling.

Cancer Genomics & ProteomicsVol. 22(1)
University Hospital Schleswig-Holstein (DE), University of Lübeck (DE)
Openalex Percentile: Top 32%
Neuroblastoma Research and Treatments
0.37
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