TEFM facilitates uterine corpus endometrial carcinoma progression by activating ROS-NFκB pathway
This study demonstrates that TEFM positively regulates autophagy to promote the growth and metastasis of UCEC cells, which provides a potential prognostic biomarker and therapeutic target for the treatment of UCEC.
Authors
- Tianjiao Lai (ORCID: https://orcid.org/0000-0002-7191-4753)
- Chunhai Fan (ORCID: https://orcid.org/0000-0002-7171-7338)
- Ruixia Guo (ORCID: https://orcid.org/0000-0003-4306-4111)
- Jiaxing Chen (ORCID: https://orcid.org/0000-0002-1818-8799)
- Mengmeng Cui (ORCID: https://orcid.org/0009-0006-5732-5174)
- Lulu Si (ORCID: https://orcid.org/0000-0002-1722-6271)
- Yana Liu (ORCID: https://orcid.org/0000-0002-7642-4219)
- Dengke Bao (ORCID: https://orcid.org/0000-0002-6503-934X)
- Yuxi Jin (ORCID: https://orcid.org/0000-0002-4344-0669)
- Qi Yang (ORCID: https://orcid.org/0000-0003-4155-0714)
- Lixia Qi
- Zhixiong Jiang
- Jianghao Guo
- Qingguo Zhu
- Jia Lei
- Lin Wang (ORCID: https://orcid.org/0000-0002-1588-588X)
Institutions
- Henan University (CN)
- First Affiliated Hospital of Henan University (CN)
- First Affiliated Hospital of Zhengzhou University (CN)
- Henan Provincial Center for Disease Control and Prevention (CN)
Publication Details
- Journal
- Journal of Translational Medicine
- Published
- 2024-12-27
- DOI
- https://doi.org/10.1186/s12967-024-05833-0
- Citations
- 2
- Primary Topic
- Mitochondrial Function and Pathology
- Type
- article
- Field-Weighted Citation Impact
- 0.47
Funders
- National Natural Science Foundation of China