Novel antileukemic compound with sub-micromolar potency against STAT5 addicted myeloid leukemia cells

Signal Transdcer and Activator of Transcription 5A and 5B (STAT5A/5B) are key effectors of tyrosine kinase oncogenes in myeloid leukemias. It is now clearly evidenced that inhibition of STAT5A/5B not only blocks the growth and survival of myeloid leukemia cells but also overcomes the resistance of leukemic cells to chemotherapy. Previous screening experiments allowed us to identify 17f as a lead compound with promising antileukemic activity that blocks the phosphorylation and transcriptional activity of STAT5A/5B in myeloid leukemia cells addicted to these proteins. In light of these findings, we initiated further pharmacomodulations of 17f to develop new derivatives with enhanced antileukemic activity. Our screening assays identified 14a, an aminopyrimidine derivative of 17f, as a new lead compound that: 1) blocks the growth and survival of myeloid leukemia cells at sub-micromolar concentrations, 2) targets the phosphorylation of STAT5 but also the expression of STAT5B and 3) relieves the resistance of Chronic and Acute Myeloid leukemia cells to conventional chemotherapy.

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Publication Details

Journal
European Journal of Medicinal Chemistry
Published
2024-12-26
DOI
https://doi.org/10.1016/j.ejmech.2024.117211
Citations
1
Primary Topic
Acute Myeloid Leukemia Research
Type
article
Field-Weighted Citation Impact
0.45
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article

Novel antileukemic compound with sub-micromolar potency against STAT5 addicted myeloid leukemia cells

Fabrice Gouilleux, Marion Polomski, Gildas Prié, Marie Brachet‐Botineau et al.
1 citations
European Journal of Medicinal Chemistry
Acute Myeloid Leukemia Research
0.45
article

Novel antileukemic compound with sub-micromolar potency against STAT5 addicted myeloid leukemia cells

Fabrice Gouilleux, Marion Polomski, Gildas Prié, Marie Brachet‐Botineau, Cécile Croix, Benjamin Victoir
article en
1 citations

Abstract

Signal Transdcer and Activator of Transcription 5A and 5B (STAT5A/5B) are key effectors of tyrosine kinase oncogenes in myeloid leukemias. It is now clearly evidenced that inhibition of STAT5A/5B not only blocks the growth and survival of myeloid leukemia cells but also overcomes the resistance of leukemic cells to chemotherapy. Previous screening experiments allowed us to identify 17f as a lead compound with promising antileukemic activity that blocks the phosphorylation and transcriptional activity of STAT5A/5B in myeloid leukemia cells addicted to these proteins. In light of these findings, we initiated further pharmacomodulations of 17f to develop new derivatives with enhanced antileukemic activity. Our screening assays identified 14a, an aminopyrimidine derivative of 17f, as a new lead compound that: 1) blocks the growth and survival of myeloid leukemia cells at sub-micromolar concentrations, 2) targets the phosphorylation of STAT5 but also the expression of STAT5B and 3) relieves the resistance of Chronic and Acute Myeloid leukemia cells to conventional chemotherapy.

European Journal of Medicinal ChemistryVol. 284
Université de Tours (FR), Inserm (FR), Physiopathologie, diagnostic et traitements des maladies musculo-squelettiques (FR)
Good health and well-being
Openalex Percentile: Top 28%
Acute Myeloid Leukemia Research
0.45
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Novel antileukemic compound with sub-micromolar potency against STAT5 addicted myeloid leukemia cells — Fabrice Gouilleux, Marion Polomski, et al. · European Journal of Medicinal Chemistry (2024) | TGRS Research Map | TGRS