Atp6v0d2 deficiency partially restores defects in Mcoln1-deficient mouse corpus luteum
This study shows that <i>Atp6v0d2</i> <sup>-<i>/</i>-</sup> <i>Mcoln1</i> <sup>-<i>/</i>-</sup> CLs had varied improvements compared with <i>Mcoln1</i> <sup>-/-</sup> CLs, and it provides <i>in vivo</i> genetic evidence of the coordination between different lysosomal channels in CL function.
Authors
- Shuo Xiao (ORCID: https://orcid.org/0000-0001-7225-9132)
- Zidao Wang (ORCID: https://orcid.org/0000-0003-4929-1817)
- Venkata Atluri (ORCID: https://orcid.org/0000-0002-8070-6745)
- Yuehuan Li (ORCID: https://orcid.org/0000-0001-8836-0198)
- Ahmed E. El Zowalaty (ORCID: https://orcid.org/0000-0001-6177-6142)
- Jaymie Bromfield
- Xiaoqin Ye
- Suvitha Viswanathan
- Jonathan Matthew Hancock
- Christian Lee Andersen (ORCID: https://orcid.org/0000-0002-4785-7533)
- Taylor Elijah Martin
- Tingjie Zhan (ORCID: https://orcid.org/0009-0004-6103-5626)
- Karly Rae Kallish
- Hope Nicole Grismer
- Yingzheng Wang (ORCID: https://orcid.org/0009-0006-1570-3624)
Institutions
- Rutgers, The State University of New Jersey (US)
- University of Georgia (US)
- Environmental and Occupational Health Sciences Institute (US)
Publication Details
- Journal
- Reproductive and Developmental Medicine
- Published
- 2024-12-11
- DOI
- https://doi.org/10.1097/rd9.0000000000000116
- Primary Topic
- Cellular transport and secretion
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- University of Georgia
- National Institutes of Health
- Office of Research on Women's Health