PRMT1-mediated methylation regulates MLL2 stability and gene expression

The interplay between multiple transcription factors precisely regulates eukaryotic transcription. Here, we report that the protein methyltransferases, MLL2/KMT2B and PRMT1, interact directly and act collectively to regulate gene expression. PRMT1 binds to the N-terminal region of MLL2, considered an intrinsically disordered region, and methylates multiple arginine residues within its RGG/RG motifs. Notably, overexpression of PRMT1 decreased poly-ubiquitylation of MLL2, whereas mutations on methylation sites in MLL2 increased MLL2 poly-ubiquitylation, suggesting that PRMT1-mediated methylation stabilizes MLL2. MLL2 and PRMT1 cooperatively stimulated the expression of a chromosomal reporter gene in a PRMT1-mediated, MLL2-methylation-dependent manner. RNA-seq analysis found that MLL2 and PRMT1 jointly regulate the expression of genes involved in cell membrane and extracellular matrix functions, and depletion of either resulted in impaired cell migration and invasion. Our study provides evidence that PRMT1-mediated MLL2 methylation regulates MLL2 protein stability and the expression of their target genes.

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Publication Details

Journal
Nucleic Acids Research
Published
2024-12-19
DOI
https://doi.org/10.1093/nar/gkae1227
Citations
5
Primary Topic
Cancer-related gene regulation
Type
article
Field-Weighted Citation Impact
1.17

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article

PRMT1-mediated methylation regulates MLL2 stability and gene expression

Hyoungmin Kim, J. Eugene Lee, Huu Phuc Nguyen, Dongju An et al.
5 citations
Nucleic Acids Research
Cancer-related gene regulation
1.17
article

PRMT1-mediated methylation regulates MLL2 stability and gene expression

Hyoungmin Kim, J. Eugene Lee, Huu Phuc Nguyen, Dongju An, Jaehoon Kim, So Yeon Park, Jung‐Ae Kim, Byul Moon, Jihyun Kim
article en
5 citations

Abstract

The interplay between multiple transcription factors precisely regulates eukaryotic transcription. Here, we report that the protein methyltransferases, MLL2/KMT2B and PRMT1, interact directly and act collectively to regulate gene expression. PRMT1 binds to the N-terminal region of MLL2, considered an intrinsically disordered region, and methylates multiple arginine residues within its RGG/RG motifs. Notably, overexpression of PRMT1 decreased poly-ubiquitylation of MLL2, whereas mutations on methylation sites in MLL2 increased MLL2 poly-ubiquitylation, suggesting that PRMT1-mediated methylation stabilizes MLL2. MLL2 and PRMT1 cooperatively stimulated the expression of a chromosomal reporter gene in a PRMT1-mediated, MLL2-methylation-dependent manner. RNA-seq analysis found that MLL2 and PRMT1 jointly regulate the expression of genes involved in cell membrane and extracellular matrix functions, and depletion of either resulted in impaired cell migration and invasion. Our study provides evidence that PRMT1-mediated MLL2 methylation regulates MLL2 protein stability and the expression of their target genes.

Nucleic Acids ResearchVol. 53(4)
Korea Advanced Institute of Science and Technology (KR), Korea Research Institute of Standards and Science (KR), Korea Research Institute of Bioscience and Biotechnology (KR), Korea University of Science and Technology (KR)
National Research Foundation, National Research Foundation of Korea
Openalex Percentile: Top 21%
Cancer-related gene regulation
1.17
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PRMT1-mediated methylation regulates MLL2 stability and gene expression — Hyoungmin Kim, J. Eugene Lee, et al. · Nucleic Acids Research (2024) | TGRS Research Map | TGRS