Dynamic Neuro-Glial-Vascular Responses in a Mouse Model of Vascular Cognitive Impairment
Together, these data suggest that, in young mice, BCAS leads to vascular dysfunction (e.g., impaired vasodilation in parenchymal arterioles), and in the absence of neuronal dysfunction, mild ischemia is associated with the activation of glial-derived neuroprotective signals.
Authors
- Weston Bush
- Zsolt Bagi (ORCID: https://orcid.org/0000-0001-8755-2980)
- Ferdinand Althammer (ORCID: https://orcid.org/0000-0001-6018-8939)
- Jessica A. Filosa (ORCID: https://orcid.org/0000-0001-7536-2039)
- Javier E. Stern
- Michael W. Brands (ORCID: https://orcid.org/0000-0002-5005-1733)
- Ki Jung Kim (ORCID: https://orcid.org/0009-0008-6901-3463)
- Juan Ramiro Diaz (ORCID: https://orcid.org/0009-0007-4925-6049)
- Philip O’Herron (ORCID: https://orcid.org/0000-0002-8137-9432)
- Randolph E. Patterson
Institutions
- Georgia State University (US)
- Augusta University (US)
Publication Details
- Journal
- Neuroglia
- Published
- 2024-12-19
- DOI
- https://doi.org/10.3390/neuroglia5040032
- Citations
- 2
- Primary Topic
- Neurological Disease Mechanisms and Treatments
- Type
- article
- Field-Weighted Citation Impact
- 0.49
Funders
- Augusta University
- National Institutes of Health
- National Institute of Neurological Disorders and Stroke