TRPC5 Promotes Intermittent Hypoxia-Induced Cardiomyocyte Injury Through Oxidative Stress
TRPC5 is associated with structural and functional cardiac damage in patients with OSA, and TRPC5 promotes IH-induced apoptosis and mitochondrial damage in cardiomyocytes through OxS. TRPC5 may be a novel target for the diagnosis and treatment of OSA-induced myocardial injury.
Authors
- Yu Li (ORCID: https://orcid.org/0000-0001-7341-5643)
- Xiaojing Sun
- Xuan Qiu (ORCID: https://orcid.org/0000-0001-9160-1757)
- Yanli Yao
- Yulan Chen (ORCID: https://orcid.org/0000-0001-6806-9897)
- Xiaoli Zhu (ORCID: https://orcid.org/0000-0003-1723-4527)
Institutions
- Xinjiang Medical University (CN)
- First Affiliated Hospital of Xinjiang Medical University (CN)
Publication Details
- Journal
- Nature and Science of Sleep
- Published
- 2024-12-01
- DOI
- https://doi.org/10.2147/nss.s494748
- Citations
- 8
- Primary Topic
- Neuroscience of respiration and sleep
- Type
- article
- Field-Weighted Citation Impact
- 2.19
Funders
- National Natural Science Foundation of China