miR-137 regulates autophagy and apoptosis in duodenal ulcer by targeting BNIP3L

miR-137 is upregulated in DU patients and contributes to ulcer progression by inhibiting BNIP3L, reducing autophagy, and promoting apoptosis. Targeting miR-137 could provide a novel therapeutic strategy for DU management.

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Publication Details

Journal
Medicine
Published
2024-12-06
DOI
https://doi.org/10.1097/md.0000000000040568
Primary Topic
MicroRNA in disease regulation
Type
article
Field-Weighted Citation Impact
0.00
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article

miR-137 regulates autophagy and apoptosis in duodenal ulcer by targeting BNIP3L

Z.Y. Pan, Li Zhang, Jing Hu
Medicine
MicroRNA in disease regulation
article

miR-137 regulates autophagy and apoptosis in duodenal ulcer by targeting BNIP3L

Z.Y. Pan, Li Zhang, Jing Hu
article en

Abstract

miR-137 is upregulated in DU patients and contributes to ulcer progression by inhibiting BNIP3L, reducing autophagy, and promoting apoptosis. Targeting miR-137 could provide a novel therapeutic strategy for DU management.

MedicineVol. 103(49)
Hebei Medical University (CN), Second Hospital of Hebei Medical University (CN)
Good health and well-being
Openalex Percentile: Top 68%
MicroRNA in disease regulation
DOIOpen Access PDFOpenAlex
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miR-137 regulates autophagy and apoptosis in duodenal ulcer by targeting BNIP3L — Z.Y. Pan, Li Zhang, et al. · Medicine (2024) | TGRS Research Map | TGRS