Exosomal <scp>ETV4</scp> Derived From <scp>M2</scp> Macrophages Induces Growth, Glycolysis and Stemness in Hepatocellular Carcinoma by UpRegulating <scp>SULT2B1</scp> Expression

ETV4 derived from M2 macrophage exosomes promoted HCC cell proliferation, glycolysis and stemness by interacting with SULT2B1, suggesting a novel insight into developing exosome-based therapy for HCC.

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Publication Details

Journal
Liver International
Published
2024-12-06
DOI
https://doi.org/10.1111/liv.16197
Citations
3
Primary Topic
Extracellular vesicles in disease
Type
article
Field-Weighted Citation Impact
0.70
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article

Exosomal ETV4 Derived From M2 Macrophages Induces Growth, Glycolysis and Stemness in Hepatocellular Carcinoma by UpRegulating SULT2B1 Expression

Zhiyan Ma, Haibin Zhong, Xinyue Chen, Feng Gao et al.
3 citations
Liver International
Extracellular vesicles in disease
0.70
article

Exosomal ETV4 Derived From M2 Macrophages Induces Growth, Glycolysis and Stemness in Hepatocellular Carcinoma by UpRegulating SULT2B1 Expression

Zhiyan Ma, Haibin Zhong, Xinyue Chen, Feng Gao, Songgang Gu, Qiaodong Xu
article en
3 citations

Abstract

ETV4 derived from M2 macrophage exosomes promoted HCC cell proliferation, glycolysis and stemness by interacting with SULT2B1, suggesting a novel insight into developing exosome-based therapy for HCC.

Liver InternationalVol. 45(4)
Shantou University (CN), Cancer Hospital of Shantou University Medical College (CN)
Openalex Percentile: Top 28%
Extracellular vesicles in disease
0.70
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