Significantly higher rates of KIDINS220 polymorphisms in patients with obesity and end-stage renal disease
Mutations within KIDINS220 may play a modulatory role in the progression of chronic kidney disease in patients with obesity, as evidenced by this small retrospective study. The relationship between KIDINS200, kidney disease, and obesity is complex and requires further study, but may represent a potential therapeutic target in the future.
Authors
- Sofia B. Ahmed (ORCID: https://orcid.org/0000-0003-3000-2229)
- Jesse Richards (ORCID: https://orcid.org/0009-0004-7985-8882)
- Madisen Fae Dorand (ORCID: https://orcid.org/0000-0003-1973-1009)
- Maria Paszkowiak
- Courtney McCorkle
- Pranay Kathuria
Institutions
- Tulsa Community College (US)
- University of Oklahoma (US)
- University of Tulsa (US)
Publication Details
- Journal
- Obesity Pillars
- Published
- 2024-12-06
- DOI
- https://doi.org/10.1016/j.obpill.2024.100155
- Citations
- 1
- Primary Topic
- Pancreatic function and diabetes
- Type
- article
- Field-Weighted Citation Impact
- 0.57
Funders
- University of Oklahoma
- College of Medicine, University of Oklahoma