[E<sub>2</sub> signaling in myofibers promots macrophage efferocytosis in mouse skeletal muscles with cardiotoxin-induced acute injury].

In injured mouse skeletal muscles, myofiber E<sub>2</sub> signaling promotes M1 to M2 transition to increase macrophage efferocytosis, thereby relieving inflammation and promoting muscle regeneration and repair.

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Publication Details

Journal
PubMed
Published
2024-11-20
DOI
https://doi.org/10.12122/j.issn.1673-4254.2024.11.16
Primary Topic
Biomedical and Chemical Research
Type
article
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article

[E2 signaling in myofibers promots macrophage efferocytosis in mouse skeletal muscles with cardiotoxin-induced acute injury].

Xiao Huang, Zonghu Liao, Hui Y. Lan, Xiaomin Niu et al.
PubMed
Biomedical and Chemical Research
article

[E2 signaling in myofibers promots macrophage efferocytosis in mouse skeletal muscles with cardiotoxin-induced acute injury].

Xiao Huang, Zonghu Liao, Hui Y. Lan, Xiaomin Niu, Bin Xian, Chao Li, Lei Liu, Na Wang, Jing Xie, Xu Hu, Qingping Cai
article en

Abstract

In injured mouse skeletal muscles, myofiber E<sub>2</sub> signaling promotes M1 to M2 transition to increase macrophage efferocytosis, thereby relieving inflammation and promoting muscle regeneration and repair.

PubMedVol. 44(11)
Southern Medical University (CN)
Biomedical and Chemical Research
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[E<sub>2</sub> signaling in myofibers promots macrophage efferocytosis in mouse skeletal muscles with cardiotoxin-induced acute injury]. — Xiao Huang, Zonghu Liao, et al. · PubMed (2024) | TGRS Research Map | TGRS