Clonal Hematopoiesis in Chronic Thromboembolic Pulmonary Hypertension

The findings in this study raise the possibility that CH will induce a more prothrombotic state through neutrophil activation and neutrophil extracellular trap formation, contributing to pathogenesis and poor treatment response in patients with CTEPH.

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Publication Details

Journal
Journal of the American Heart Association
Published
2024-11-27
DOI
https://doi.org/10.1161/jaha.124.035498
Citations
5
Primary Topic
Eosinophilic Disorders and Syndromes
Type
article
Field-Weighted Citation Impact
2.35
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article

Clonal Hematopoiesis in Chronic Thromboembolic Pulmonary Hypertension

Jin Endo, Hideaki Nakajima, Masaki Ieda, Yoshinori Katsumata et al.
5 citations
Journal of the American Heart Association
Eosinophilic Disorders and Syndromes
2.35
article

Clonal Hematopoiesis in Chronic Thromboembolic Pulmonary Hypertension

Jin Endo, Hideaki Nakajima, Masaki Ieda, Yoshinori Katsumata, Kohsuke Shirakawa, Yoshiki Shinya, Hiroyoshi Kunimoto, Takumi Inami, Fuyuki Miya, Mizuki Momoi, Keiichi Fukuda, Takahiro Hiraide, Masaharu Kataoka, Shinichi Goto, Kenjiro Kosaki, Atsushi Anzai, Ayaka Miura
article en
5 citations

Abstract

The findings in this study raise the possibility that CH will induce a more prothrombotic state through neutrophil activation and neutrophil extracellular trap formation, contributing to pathogenesis and poor treatment response in patients with CTEPH.

Journal of the American Heart AssociationVol. 13(23)
Brigham and Women's Hospital (US), Tokai University (JP), Harvard University (US), University of Occupational and Environmental Health Japan (JP), Kyorin University (JP), Keio University (JP), Yokohama City University (JP)
Good health and well-being
Openalex Percentile: Top 11%
Eosinophilic Disorders and Syndromes
2.35
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Clonal Hematopoiesis in Chronic Thromboembolic Pulmonary Hypertension — Jin Endo, Hideaki Nakajima, et al. · Journal of the American Heart Association (2024) | TGRS Research Map | TGRS