Exploring the role of oxidative stress and mitochondrial dysfunction in β-damascone-induced aneuploidy
These findings suggest that the oxidative stress induced by β-Dam exposure may be explained through an aneugenic MoA via mitochondrial dysfunction, thereby contributing to MN formation in mammalian cells.
Authors
- Tsuneo Hashizume (ORCID: https://orcid.org/0000-0001-7595-5440)
- Satoru Munakata
- Taku Watanabe (ORCID: https://orcid.org/0000-0002-3195-2413)
- Tomohiro Takahashi
Institutions
- Japan Tobacco (United States) (US)
- Japan Tobacco (Japan) (JP)
Publication Details
- Journal
- Genes and Environment
- Published
- 2024-11-25
- DOI
- https://doi.org/10.1186/s41021-024-00319-3
- Primary Topic
- Plant Disease Resistance and Genetics
- Type
- article
- Field-Weighted Citation Impact
- 0.00