Chronic inflammation degrades CD4 T cell immunity to prior vaccines in treated HIV infection

To date, our understanding of how HIV infection impacts vaccine-induced cellular immunity is limited. Here, we investigate inflammation, immune activation and antigen-specific T cell responses in HIV-uninfected and antiretroviral-treated HIV-infected people. Our findings highlight lower recall responses of antigen-specific CD4 T cells that correlate with high plasma cytokines levels, T cell hyperactivation and an altered composition of the T subsets enriched with more differentiated cells in the HIV-infected group. Transcriptomic analysis reveals that antigen-specific CD4 T cells of the HIV-infected group have a reduced expression of gene sets previously reported to correlate with vaccine-induced pathogen-specific protective immunity and further identifies a consistent impairment of the IFNα and IFNγ response pathways as mechanism for the functional loss of recall CD4 T cell responses in antiretroviral-treated people. Lastly, in vitro treatment with drugs that reduce inflammation results in higher memory CD4 T cell IFNγ responses. Together, our findings suggest that vaccine-induced cellular immunity may benefit from strategies to counteract inflammation in HIV infection.

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Publication Details

Journal
Nature Communications
Published
2024-11-25
DOI
https://doi.org/10.1038/s41467-024-54605-3
Citations
7
Primary Topic
HIV Research and Treatment
Type
article
Field-Weighted Citation Impact
2.06

Funders

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article

Chronic inflammation degrades CD4 T cell immunity to prior vaccines in treated HIV infection

Farida Laboune, Ellen G. Harrer, Daniel C. Douek, Slim Fourati et al.
7 citations
Nature Communications
HIV Research and Treatment
2.06
article

Chronic inflammation degrades CD4 T cell immunity to prior vaccines in treated HIV infection

Farida Laboune, Ellen G. Harrer, Daniel C. Douek, Slim Fourati, Melissa Kießling, Sabrina Kübel, Krystelle Nganou‐Makamdop, Amy R. Henry, Paulina Klein, Thomas Harrer, Klaus Korn, Klaus Überla, John J Cole
article en
7 citations

Abstract

To date, our understanding of how HIV infection impacts vaccine-induced cellular immunity is limited. Here, we investigate inflammation, immune activation and antigen-specific T cell responses in HIV-uninfected and antiretroviral-treated HIV-infected people. Our findings highlight lower recall responses of antigen-specific CD4 T cells that correlate with high plasma cytokines levels, T cell hyperactivation and an altered composition of the T subsets enriched with more differentiated cells in the HIV-infected group. Transcriptomic analysis reveals that antigen-specific CD4 T cells of the HIV-infected group have a reduced expression of gene sets previously reported to correlate with vaccine-induced pathogen-specific protective immunity and further identifies a consistent impairment of the IFNα and IFNγ response pathways as mechanism for the functional loss of recall CD4 T cell responses in antiretroviral-treated people. Lastly, in vitro treatment with drugs that reduce inflammation results in higher memory CD4 T cell IFNγ responses. Together, our findings suggest that vaccine-induced cellular immunity may benefit from strategies to counteract inflammation in HIV infection.

Nature CommunicationsVol. 15(1)
Northwestern University (US), National Institutes of Health (US), Friedrich-Alexander-Universität Erlangen-Nürnberg (DE), Universitätsklinikum Erlangen (DE), University of Glasgow (GB)
Deutsche Forschungsgemeinschaft
Good health and well-being
Openalex Percentile: Top 13%
HIV Research and Treatment
2.06
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