miR-424/322 attenuates cardiac remodeling by modulating the nuclear factor-activated T-cell 3/furin pathway
The NFATc3/miR-424/322/furin axis is crucial for developing cardiac remodeling, and exogenous miR-322 mimics could have therapeutic potential in cardiac remodeling.
Authors
- Shih‐Hung Tsai (ORCID: https://orcid.org/0000-0003-0215-3193)
- Shang‐Feng Yang (ORCID: https://orcid.org/0000-0002-0077-7130)
- Hsiao‐Ya Tsai (ORCID: https://orcid.org/0000-0002-5704-507X)
- Chih-Yuan Lin (ORCID: https://orcid.org/0000-0002-2321-6053)
- Po‐Hsun Huang (ORCID: https://orcid.org/0000-0003-1584-6749)
- Min‐Chien Tsai (ORCID: https://orcid.org/0000-0002-2869-2382)
- Jen‐Chun Wang (ORCID: https://orcid.org/0000-0002-1661-888X)
- Chin‐Wang Hsu
- Yu-Juei Hsu
Institutions
- Tri-Service General Hospital (TW)
- Taipei Medical University Hospital (TW)
- Taipei Veterans General Hospital (TW)
- Cheng Hsin General Hospital (TW)
- Taipei Medical University (TW)
- National Defense Medical Center (TW)
Publication Details
- Journal
- Biomedical Journal
- Published
- 2024-11-23
- DOI
- https://doi.org/10.1016/j.bj.2024.100818
- Citations
- 4
- Primary Topic
- Cardiac Fibrosis and Remodeling
- Type
- article
- Field-Weighted Citation Impact
- 1.61
Funders
- Cheng Hsin General Hospital Foundation
- Tri-Service General Hospital
- National Science and Technology Council
- Medical Affairs Bureau