S100A1ct: A Synthetic Peptide Derived From S100A1 Protein Improves Cardiac Performance and Survival in Preclinical Heart Failure Models

S100A1ct is a promising lead for the development of novel peptide-based therapeutics against heart failure with reduced ejection fraction.

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Publication Details

Journal
Circulation
Published
2024-11-21
DOI
https://doi.org/10.1161/circulationaha.123.066961
Citations
6
Primary Topic
S100 Proteins and Annexins
Type
article
Field-Weighted Citation Impact
1.40
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article

S100A1ct: A Synthetic Peptide Derived From S100A1 Protein Improves Cardiac Performance and Survival in Preclinical Heart Failure Models

Rebecca C. Wade, Julia Ritterhoff, Norbert Frey, Jennifer Birkenstock et al.
6 citations
Circulation
S100 Proteins and Annexins
1.40
article

S100A1ct: A Synthetic Peptide Derived From S100A1 Protein Improves Cardiac Performance and Survival in Preclinical Heart Failure Models

Rebecca C. Wade, Julia Ritterhoff, Norbert Frey, Jennifer Birkenstock, Kristin Spaich, Mirko Völkers, Manuel Glaser, Arjang Ruhparwar, Dorothea Kehr, Erhe Gao, Paul Mather, Hugo A. Katus, Cornelius Busch, Andreas Jungmann, Patrick Most, Michael E. Egger, Karl Varadi, Lukas Jarosch, Walter J. Koch, Marc Freichel, Rafael Salazar, Max Sauter, Martin Busch
article en
6 citations

Abstract

S100A1ct is a promising lead for the development of novel peptide-based therapeutics against heart failure with reduced ejection fraction.

CirculationVol. 151(8)
Thomas Jefferson University (US), Duke University (US), Heidelberg University (DE), University Hospital Heidelberg (DE), Medizinische Hochschule Hannover (DE), Max Planck Institute for Medical Research (DE), Kraftanlagen (Germany) (DE), German Centre for Cardiovascular Research (DE), Computing Center (RU), DKFZ-ZMBH Alliance (DE), Heidelberg Institute for Theoretical Studies (DE), University of Pennsylvania (US), Temple University (US)
Good health and well-being
Openalex Percentile: Top 18%
S100 Proteins and Annexins
1.40
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