Slow Subcutaneous Release of Glatiramer Acetate or CD40-Targeting Peptide KGYY6 Is More Advantageous in Treating Ongoing Experimental Autoimmune Encephalomyelitis

Slow-release formulations of both GA and KGYY<sub>6</sub> vastly improve the efficacy of both, and KGYY<sub>6</sub> is more consistent in efficacy across different lots.

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Publication Details

Journal
Neurology International
Published
2024-11-20
DOI
https://doi.org/10.3390/neurolint16060114
Primary Topic
Immunotherapy and Immune Responses
Type
article
Field-Weighted Citation Impact
0.00

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article

Slow Subcutaneous Release of Glatiramer Acetate or CD40-Targeting Peptide KGYY6 Is More Advantageous in Treating Ongoing Experimental Autoimmune Encephalomyelitis

David H. Wagner, Gisela M. Vaitaitis
Neurology International
Immunotherapy and Immune Responses
article

Slow Subcutaneous Release of Glatiramer Acetate or CD40-Targeting Peptide KGYY6 Is More Advantageous in Treating Ongoing Experimental Autoimmune Encephalomyelitis

David H. Wagner, Gisela M. Vaitaitis
article en

Abstract

Slow-release formulations of both GA and KGYY<sub>6</sub> vastly improve the efficacy of both, and KGYY<sub>6</sub> is more consistent in efficacy across different lots.

Neurology InternationalVol. 16(6)
University of Colorado Anschutz Medical Campus (US)
National Institute of Diabetes and Digestive and Kidney Diseases
Good health and well-being
Openalex Percentile: Top 73%
Immunotherapy and Immune Responses
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Slow Subcutaneous Release of Glatiramer Acetate or CD40-Targeting Peptide KGYY6 Is More Advantageous in Treating Ongoing Experimental Autoimmune Encephalomyelitis — David H. Wagner, Gisela M. Vaitaitis · Neurology International (2024) | TGRS Research Map | TGRS