GRP78 mediates mitochondrial fusion and fission in cigarette smoke-induced inflammatory responses in airway epithelial cells
Taken together, these results suggested that the GRP78-mediated mitochondrial fusion and fission process played a vital role in cigarette smoke-induced airway inflammation and might be a potential therapeutic target in this regard.
Authors
- Fei Gao (ORCID: https://orcid.org/0000-0002-4347-8499)
- Yong Wang (ORCID: https://orcid.org/0000-0002-0086-0882)
- Chenchen Li (ORCID: https://orcid.org/0000-0002-8047-186X)
- Yajing Li (ORCID: https://orcid.org/0009-0001-6296-7394)
- Wei Geng (ORCID: https://orcid.org/0000-0002-7999-7581)
- Li Jin Pu
- Qing Zhang (ORCID: https://orcid.org/0000-0002-9987-5661)
Institutions
- Anhui Medical University (CN)
- First Affiliated Hospital of Anhui Medical University (CN)
Publication Details
- Journal
- Inhalation Toxicology
- Published
- 2024-11-20
- DOI
- https://doi.org/10.1080/08958378.2024.2428163
- Primary Topic
- Mitochondrial Function and Pathology
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Natural Science Foundation of China