miR-361-3p overexpression promotes apoptosis and inflammation by regulating the USP49/IκBα/NF-κB pathway to aggravate sepsis-induced myocardial injury
Taken together, our data demonstrate that miR-361-3p overexpression promotes apoptosis and inflammation by regulating the USP49/IκBα/NF-κB pathway to aggravate sepsis-induced myocardial injury.
Authors
- Lijiao You (ORCID: https://orcid.org/0000-0003-0957-0109)
- Hongzhi Geng (ORCID: https://orcid.org/0000-0003-0788-3734)
- Luyao Qi (ORCID: https://orcid.org/0009-0005-3723-7452)
- Xiaofang Yang (ORCID: https://orcid.org/0000-0001-9341-2378)
- Wentao Feng (ORCID: https://orcid.org/0009-0003-7543-9827)
- Ming Lei (ORCID: https://orcid.org/0000-0001-5558-1285)
Institutions
- Shanghai University of Traditional Chinese Medicine (CN)
- Seventh People's Hospital of Shanghai (CN)
Publication Details
- Journal
- Toxicology Research
- Published
- 2024-11-05
- DOI
- https://doi.org/10.1093/toxres/tfae190
- Citations
- 2
- Primary Topic
- MicroRNA in disease regulation
- Type
- article
- Field-Weighted Citation Impact
- 0.57
Funders
- National Natural Science Foundation of China
- Hainan Medical University
- Shanghai Pudong New Area Health Commission