Stochastic differential equations model for the interaction of HCV with liver cells

The main objective of this paper is to study the dynamic behavior of the hepatitis C virus to control the spread of this disease. The study was conducted by creating a new stochastic mathematical model. Three equilibriums specific to this system are discussed, namely, disease-free equilibrium, equilibrium in the absence of an antibody response, and equilibrium when immune response CTLs are zero. We also showed conditions that must be met for the injured person to recover by searching for the basic reproduction number. If it means that the liver will get rid of the virus and heal the infected person. while in this case, the infection grows and the disease can invade all liver cells. These results have also been demonstrated by computer simulations.

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Publication Details

Journal
Academic Science Journal
Published
2024-10-01
DOI
https://doi.org/10.24237/asj.02.04.794b
Citations
1
Primary Topic
Mathematical and Theoretical Epidemiology and Ecology Models
Type
article
Field-Weighted Citation Impact
0.54
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article

Stochastic differential equations model for the interaction of HCV with liver cells

Ahmed Kareem, Faris J.Wafar
1 citations
Academic Science Journal
Mathematical and Theoretical Epidemiology and Ecology Models
0.54
article

Stochastic differential equations model for the interaction of HCV with liver cells

Ahmed Kareem, Faris J.Wafar
article en
1 citations

Abstract

The main objective of this paper is to study the dynamic behavior of the hepatitis C virus to control the spread of this disease. The study was conducted by creating a new stochastic mathematical model. Three equilibriums specific to this system are discussed, namely, disease-free equilibrium, equilibrium in the absence of an antibody response, and equilibrium when immune response CTLs are zero. We also showed conditions that must be met for the injured person to recover by searching for the basic reproduction number. If it means that the liver will get rid of the virus and heal the infected person. while in this case, the infection grows and the disease can invade all liver cells. These results have also been demonstrated by computer simulations.

Academic Science JournalVol. 2(4)
University of Diyala (IQ)
Openalex Percentile: Top 27%
Mathematical and Theoretical Epidemiology and Ecology Models
0.54
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