Identification of PDLIM1 as a glioblastoma stem cell marker driving tumorigenesis and chemoresistance

Glioblastoma (GBM) is an aggressive brain tumor with a poor prognosis, largely due to the presence of glioblastoma stem cells (GSCs). These cells drive tumor progression, recurrence, and chemoresistance, making them critical targets for therapy. This study aims to identify novel GSC markers for improved diagnosis and targeted treatment. We utilized single-cell RNA sequencing (scRNA-seq) and bulk RNA-seq data to identify PDLIM1 as a novel GSC marker. PDLIM1 was specifically expressed in GSCs and was associated with poor prognosis and advanced tumor stages. Functional assays demonstrated that PDLIM1 overexpression enhanced GBM cell proliferation, reduced apoptosis, increased GSC proportions, and promoted chemoresistance and tumorigenesis. Conversely, PDLIM1 knockdown inhibited these processes. Mechanistically, PDLIM1 was found to exert its effects likely by promoting the PI3K-AKT pathway. In conclusion, PDLIM1 may serve as a potential marker of GSCs associated with poor prognosis, tumorigenesis, and chemoresistance in GBM, representing a potential therapeutic target for improving GBM patient outcomes.

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Publication Details

Journal
Cell Death Discovery
Published
2024-11-15
DOI
https://doi.org/10.1038/s41420-024-02241-7
Citations
4
Primary Topic
Glioma Diagnosis and Treatment
Type
article
Field-Weighted Citation Impact
1.75

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article

Identification of PDLIM1 as a glioblastoma stem cell marker driving tumorigenesis and chemoresistance

Yunfeng Liu, Xiaopeng Shen, Jian Ruan, Yun Fu Zhao et al.
4 citations
Cell Death Discovery
Glioma Diagnosis and Treatment
1.75
article

Identification of PDLIM1 as a glioblastoma stem cell marker driving tumorigenesis and chemoresistance

Yunfeng Liu, Xiaopeng Shen, Jian Ruan, Yun Fu Zhao, Shan Lu, Guoping Zhu, Meng Li, Yang Cao, Huaizhang Jin, Chunguang Wang
article en
4 citations

Abstract

Glioblastoma (GBM) is an aggressive brain tumor with a poor prognosis, largely due to the presence of glioblastoma stem cells (GSCs). These cells drive tumor progression, recurrence, and chemoresistance, making them critical targets for therapy. This study aims to identify novel GSC markers for improved diagnosis and targeted treatment. We utilized single-cell RNA sequencing (scRNA-seq) and bulk RNA-seq data to identify PDLIM1 as a novel GSC marker. PDLIM1 was specifically expressed in GSCs and was associated with poor prognosis and advanced tumor stages. Functional assays demonstrated that PDLIM1 overexpression enhanced GBM cell proliferation, reduced apoptosis, increased GSC proportions, and promoted chemoresistance and tumorigenesis. Conversely, PDLIM1 knockdown inhibited these processes. Mechanistically, PDLIM1 was found to exert its effects likely by promoting the PI3K-AKT pathway. In conclusion, PDLIM1 may serve as a potential marker of GSCs associated with poor prognosis, tumorigenesis, and chemoresistance in GBM, representing a potential therapeutic target for improving GBM patient outcomes.

Cell Death DiscoveryVol. 10(1)
Wannan Medical College (CN), First Affiliated Hospital of Wannan Medical College (CN), Anhui Normal University (CN)
Natural Science Foundation of Anhui Province
No poverty
Openalex Percentile: Top 14%
Glioma Diagnosis and Treatment
1.75
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Identification of PDLIM1 as a glioblastoma stem cell marker driving tumorigenesis and chemoresistance — Yunfeng Liu, Xiaopeng Shen, et al. · Cell Death Discovery (2024) | TGRS Research Map | TGRS