FOXN3 Regulates Autophagic Activity to Suppress Drug Resistance in Melanoma Cells
FOXN3 functions as an important regulator of the development and progression of Vemurafenib-resistant melanoma cells, partly owing to its binding to the <i>FISP1</i>. As such, FOXN3 may represent a relevant target for therapeutic interventions in patients suffering from drug-resistant melanoma.
Authors
- Tong Xiao (ORCID: https://orcid.org/0000-0003-1462-4650)
- Chen Tu (ORCID: https://orcid.org/0000-0002-7777-4413)
- Yaqi Wang (ORCID: https://orcid.org/0000-0002-3361-440X)
- Hui Su (ORCID: https://orcid.org/0009-0006-0884-6301)
- Bincheng Ren
- Xiaopeng Wang (ORCID: https://orcid.org/0000-0002-7036-1616)
- Shuang Wang (ORCID: https://orcid.org/0009-0007-1593-258X)
Institutions
- Second Affiliated Hospital of Xi'an Jiaotong University (CN)
- Second Affiliated Hospital of Zhejiang University (CN)
Publication Details
- Journal
- Clinical Cosmetic and Investigational Dermatology
- Published
- 2024-11-01
- DOI
- https://doi.org/10.2147/ccid.s462854
- Primary Topic
- Autophagy in Disease and Therapy
- Type
- article
- Field-Weighted Citation Impact
- 0.00