Polygenic Risk of Epilepsy and Poststroke Epilepsy

Our findings suggest that, like other forms of epilepsy, genetic predisposition plays an essential role in PSE. These results underscore the need for future studies to elucidate the mechanisms underlying PSE development and to identify novel therapeutic avenues.

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Publication Details

Journal
Stroke
Published
2024-11-06
DOI
https://doi.org/10.1161/strokeaha.124.047459
Citations
14
Primary Topic
Pharmacological Effects and Toxicity Studies
Type
article
Field-Weighted Citation Impact
15.53

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article

Polygenic Risk of Epilepsy and Poststroke Epilepsy

Jennifer A. Kim, Rajarshi Mazumder, Lauren Hachmann Sansing, Emily J. Gilmore et al.
14 citations
Stroke
Pharmacological Effects and Toxicity Studies
15.53
article

Polygenic Risk of Epilepsy and Poststroke Epilepsy

Jennifer A. Kim, Rajarshi Mazumder, Lauren Hachmann Sansing, Emily J. Gilmore, David S. Liebeskind, Cyprien Rivier, Johan Zelano, Guido J. Falcone, Adam de Havenon, Kevin Navin Sheth, Santiago Clocchiatti‐Tuozzo, Nishant K. Mishra, Shubham Misra, Lawrence J. Hirsch, Bradford B. Worrall
article en
14 citations

Abstract

Our findings suggest that, like other forms of epilepsy, genetic predisposition plays an essential role in PSE. These results underscore the need for future studies to elucidate the mechanisms underlying PSE development and to identify novel therapeutic avenues.

StrokeVol. 55(12)
University of California, Los Angeles (US), VA Connecticut Research and Education Foundation (US), University of Virginia (US), University of Gothenburg (SE)
Yale University, GW Pharmaceuticals, SK Life Science, Eisai, Novo Nordisk, Sahlgrenska Universitetssjukhuset, National Institutes of Health, Genentech, Fogarty International Center
Openalex Percentile: Top 1%
Pharmacological Effects and Toxicity Studies
15.53
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