A hypothesis to explain malaria-induced neurocognitive sequelae
The development of malaria-induced neurocognitive and behavioral sequelae is not entirely understood. We hypothesize that liver dysfunction caused by Plasmodium infection is responsible for malaria-induced neurocognitive and behavioral sequelae. Our metabolic hypothesis not only explains neurocognitive sequelae after cerebral malaria (CM) but also after other severe, non-severe, and asymptomatic malaria infections.
Authors
- Cláudio Tadeu Daniel‐Ribeiro (ORCID: https://orcid.org/0000-0001-9075-1470)
- Yuri Chaves Martins (ORCID: https://orcid.org/0000-0003-4220-4979)
Institutions
- Saint Louis University (ES)
- Saint Louis University (US)
- Fundação Oswaldo Cruz (BR)
Publication Details
- Journal
- Trends in Parasitology
- Published
- 2024-11-04
- DOI
- https://doi.org/10.1016/j.pt.2024.10.008
- Citations
- 4
- Primary Topic
- Malaria Research and Control
- Type
- article
- Field-Weighted Citation Impact
- 2.17
Funders
- Saint Louis University
- Conselho Nacional de Desenvolvimento Científico e Tecnológico