A multiplex single-cell RNA-Seq pharmacotranscriptomics pipeline for drug discovery

The gene-regulatory dynamics governing drug responses in cancer are yet to be fully understood. Here, we report a pipeline capable of producing high-throughput pharmacotranscriptomic profiling through live-cell barcoding using antibody-oligonucleotide conjugates. This pipeline combines drug screening with 96-plex single-cell RNA sequencing. We show the potential of this approach by exploring the heterogeneous transcriptional landscape of primary high-grade serous ovarian cancer (HGSOC) cells after treatment with 45 drugs, with 13 distinct classes of mechanisms of action. A subset of phosphatidylinositol 3-OH kinase (PI3K), protein kinase B (AKT) and mammalian target of rapamycin (mTOR) inhibitors induced the activation of receptor tyrosine kinases, such as the epithelial growth factor receptor (EGFR), and this was mediated by the upregulation of caveolin 1 (CAV1). This drug resistance feedback loop could be mitigated by the synergistic action of agents targeting PI3K-AKT-mTOR and EGFR for HGSOC with CAV1 and EGFR expression. Using this workflow could enable the personalized testing of patient-derived tumor samples at single-cell resolution.

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Publication Details

Journal
Nature Chemical Biology
Published
2024-10-31
DOI
https://doi.org/10.1038/s41589-024-01761-8
Citations
18
Primary Topic
Cancer Genomics and Diagnostics
Type
article
Field-Weighted Citation Impact
5.13

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article

A multiplex single-cell RNA-Seq pharmacotranscriptomics pipeline for drug discovery

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18 citations
Nature Chemical Biology
Cancer Genomics and Diagnostics
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article

A multiplex single-cell RNA-Seq pharmacotranscriptomics pipeline for drug discovery

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article en
18 citations

Abstract

The gene-regulatory dynamics governing drug responses in cancer are yet to be fully understood. Here, we report a pipeline capable of producing high-throughput pharmacotranscriptomic profiling through live-cell barcoding using antibody-oligonucleotide conjugates. This pipeline combines drug screening with 96-plex single-cell RNA sequencing. We show the potential of this approach by exploring the heterogeneous transcriptional landscape of primary high-grade serous ovarian cancer (HGSOC) cells after treatment with 45 drugs, with 13 distinct classes of mechanisms of action. A subset of phosphatidylinositol 3-OH kinase (PI3K), protein kinase B (AKT) and mammalian target of rapamycin (mTOR) inhibitors induced the activation of receptor tyrosine kinases, such as the epithelial growth factor receptor (EGFR), and this was mediated by the upregulation of caveolin 1 (CAV1). This drug resistance feedback loop could be mitigated by the synergistic action of agents targeting PI3K-AKT-mTOR and EGFR for HGSOC with CAV1 and EGFR expression. Using this workflow could enable the personalized testing of patient-derived tumor samples at single-cell resolution.

Nature Chemical BiologyVol. 21(3)
University of Helsinki (FI), Tampere University Hospital (FI), Tampere University (FI), Institute for Molecular Medicine Finland (FI), University of Oulu (FI)
Helsingin Yliopisto, Academy of Finland, China Scholarship Council, Oulun Yliopisto, Sigrid Juséliuksen Säätiö, Syöpäsäätiö, Biocenter Finland, Helsinki Institute of Life Science, Helsingin Yliopisto
Good health and well-being
Openalex Percentile: Top 4%
Cancer Genomics and Diagnostics
5.13
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