Kiss1 receptor knockout exacerbates airway hyperresponsiveness and remodeling in a mouse model of allergic asthma

These findings demonstrate the importance of inherent Kiss1R signaling in regulating airway inflammation, AHR, and remodeling in the pathophysiology of asthma.

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Publication Details

Journal
Respiratory Research
Published
2024-10-28
DOI
https://doi.org/10.1186/s12931-024-03017-4
Citations
4
Primary Topic
Hypothalamic control of reproductive hormones
Type
article
Field-Weighted Citation Impact
2.15

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article

Kiss1 receptor knockout exacerbates airway hyperresponsiveness and remodeling in a mouse model of allergic asthma

Nilesh Sudhakar Ambhore, Venkatachalem Sathish, Jacob Tesch, Sahil Lohana et al.
4 citations
Respiratory Research
Hypothalamic control of reproductive hormones
2.15
article

Kiss1 receptor knockout exacerbates airway hyperresponsiveness and remodeling in a mouse model of allergic asthma

Nilesh Sudhakar Ambhore, Venkatachalem Sathish, Jacob Tesch, Sahil Lohana, Mohammad Irshad Reza, Y. Ramakrishnan, Ashish Kumar, Premanand Balraj
article en
4 citations

Abstract

These findings demonstrate the importance of inherent Kiss1R signaling in regulating airway inflammation, AHR, and remodeling in the pathophysiology of asthma.

Respiratory ResearchVol. 25(1)
North Dakota State University (US)
National Heart, Lung, and Blood Institute, National Institute of General Medical Sciences
Good health and well-being
Openalex Percentile: Top 12%
Hypothalamic control of reproductive hormones
2.15
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Kiss1 receptor knockout exacerbates airway hyperresponsiveness and remodeling in a mouse model of allergic asthma — Nilesh Sudhakar Ambhore, Venkatachalem Sathish, et al. · Respiratory Research (2024) | TGRS Research Map | TGRS