The molecular mechanisms by which the NLRP3 inflammasome regulates blood-brain barrier permeability following cryptococcal meningitis

Vimentin and the NLRP3 inflammasome are both implicated in the pathological process of cryptococcal meningitis. An interaction between Vimentin and the NLRP3 inflammasome is evident, likely mediated through the NF-κB signaling pathway.

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Publication Details

Journal
Heliyon
Published
2024-10-23
DOI
https://doi.org/10.1016/j.heliyon.2024.e39653
Primary Topic
Fungal Infections and Studies
Type
article
Field-Weighted Citation Impact
0.00

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article

The molecular mechanisms by which the NLRP3 inflammasome regulates blood-brain barrier permeability following cryptococcal meningitis

Ding-An Mao, Xing‐Fang Li, Liqun Liu, Peng Huang et al.
Heliyon
Fungal Infections and Studies
article

The molecular mechanisms by which the NLRP3 inflammasome regulates blood-brain barrier permeability following cryptococcal meningitis

Ding-An Mao, Xing‐Fang Li, Liqun Liu, Peng Huang, Jie Xiong, Yufen Tang, Lingjuan Liu, Yangyang Xiao, Lu Zhang
article en

Abstract

Vimentin and the NLRP3 inflammasome are both implicated in the pathological process of cryptococcal meningitis. An interaction between Vimentin and the NLRP3 inflammasome is evident, likely mediated through the NF-κB signaling pathway.

HeliyonVol. 10(23)
Central South University (CN), Second Xiangya Hospital of Central South University (CN)
National Natural Science Foundation of China, Natural Science Foundation of Hunan Province, Changchun Science and Technology Planning Project
Good health and well-being
Openalex Percentile: Top 72%
Fungal Infections and Studies
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