Sulforaphane regulates AngII-induced podocyte oxidative stress injury through the Nrf2-Keap1/ho-1/ROS pathway

Sulforaphane alleviated AngII-induced podocyte oxidative stress injury <i>via</i> the Nrf2-Keap1/HO-1/ROS pathway, providing new insights into therapeutic compounds for mitigating chronic kidney disease.

Authors

Wen Lu (ORCID: https://orcid.org/0000-0001-6964-1947)

Institutions

People’s Hospital of Rizhao (CN)

Publication Details

Journal: Renal Failure
Published: 2024-10-17
DOI: https://doi.org/10.1080/0886022x.2024.2416937
Citations: 8
Primary Topic: Genomics, phytochemicals, and oxidative stress
Type: article
Field-Weighted Citation Impact: 1.87

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article

Sulforaphane regulates AngII-induced podocyte oxidative stress injury through the Nrf2-Keap1/ho-1/ROS pathway

Wen Lu

Oct 17, 2024

8 citations

Renal Failure

Genomics, phytochemicals, and oxidative stress

1.87

article

Sulforaphane regulates AngII-induced podocyte oxidative stress injury through the Nrf2-Keap1/ho-1/ROS pathway

Wen Lu

October 17, 2024article en

8 citations

Abstract

Renal FailureVol. 46(2)

People’s Hospital of Rizhao (CN)

Openalex Percentile: Top 14%

Genomics, phytochemicals, and oxidative stress

1.87

DOI Open Access PDF OpenAlex

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Sulforaphane regulates AngII-induced podocyte oxidative stress injury through the Nrf2-Keap1/ho-1/ROS pathway — Wen Lu · Renal Failure (2024) | TGRS Research Map | TGRS