BCAT1 contributes to the development of TKI-resistant CML
These findings demonstrate the role of BCAA/BCAT1 signaling in cancer development and suggest that targeting BCAA/BCAT1 signaling is a potential strategy for interfering with TKI-resistant CML.
Authors
- Li Xie (ORCID: https://orcid.org/0000-0002-8883-0943)
- Ligen Liu (ORCID: https://orcid.org/0000-0002-3971-783X)
- Dan Ding Huang (ORCID: https://orcid.org/0000-0001-8651-0471)
- Xiaoxiao He (ORCID: https://orcid.org/0000-0003-1219-1986)
- Yu Jiang (ORCID: https://orcid.org/0000-0002-6172-1130)
- Junke Zheng (ORCID: https://orcid.org/0000-0002-7023-2400)
- Chiqi Chen (ORCID: https://orcid.org/0000-0001-7220-8212)
- Ya‐Ping Zhang (ORCID: https://orcid.org/0000-0002-5401-1114)
- Zhuo Yu
- Difan Zhang
Institutions
- Shanghai Jiao Tong University (CN)
- Chinese Academy of Sciences (CN)
- Zhejiang Cancer Hospital (CN)
- Tongren Hospital (CN)
- Ministry of Education (TH)
Publication Details
- Journal
- Cellular Oncology
- Published
- 2024-10-16
- DOI
- https://doi.org/10.1007/s13402-024-01003-y
- Citations
- 7
- Primary Topic
- Chronic Myeloid Leukemia Treatments
- Type
- article
- Field-Weighted Citation Impact
- 3.18
Funders
- National Natural Science Foundation of China
- National Key Research and Development Program of China