HDAC6 promotes inflammation in lupus nephritis mice by regulating transcription factors MAFF and KLF5 in renal fibrosis

HDAC6 suppresses MAFF expression <i>via</i> deacetylation to elevate KLF5 expression, which consequently enhances fibrosis and inflammatory response in LN.

Authors

Shanshan Tao
Rui Fu (ORCID: https://orcid.org/0000-0002-0577-2830)
Meihui Deng (ORCID: https://orcid.org/0000-0001-9762-1361)
Weimin Zheng
Xiao Tan
Xiaojie Peng

Institutions

Jiangxi Provincial Children's Hospital (CN)

Publication Details

Journal: Renal Failure
Published: 2024-10-16
DOI: https://doi.org/10.1080/0886022x.2024.2415517
Citations: 8
Primary Topic: Kruppel-like factors research
Type: article
Field-Weighted Citation Impact: 1.87

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article

HDAC6 promotes inflammation in lupus nephritis mice by regulating transcription factors MAFF and KLF5 in renal fibrosis

Shanshan Tao, Rui Fu, Meihui Deng, Weimin Zheng et al.

Oct 16, 2024

8 citations

Renal Failure

Kruppel-like factors research

1.87

article

HDAC6 promotes inflammation in lupus nephritis mice by regulating transcription factors MAFF and KLF5 in renal fibrosis

Shanshan Tao, Rui Fu, Meihui Deng, Weimin Zheng, Xiao Tan, Xiaojie Peng

October 16, 2024article en

8 citations

Abstract

HDAC6 suppresses MAFF expression <i>via</i> deacetylation to elevate KLF5 expression, which consequently enhances fibrosis and inflammatory response in LN.

Renal FailureVol. 46(2)

Jiangxi Provincial Children's Hospital (CN)

Good health and well-being

Openalex Percentile: Top 14%

Kruppel-like factors research

1.87

DOI Open Access PDF OpenAlex

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HDAC6 promotes inflammation in lupus nephritis mice by regulating transcription factors MAFF and KLF5 in renal fibrosis — Shanshan Tao, Rui Fu, et al. · Renal Failure (2024) | TGRS Research Map | TGRS