Fecal Microbiota Transplantation Induced by Wumei Pills Improves Chemotherapy-Induced Intestinal Mucositis in BALB/c Mice by Modulating the TLR4/MyD88/NF-κB Signaling Pathway

FMT can defend the intestinal mucosal barrier integrality by increasing the content of exercise fatty acids, and its mechanism may be in connection with its inhibition of TLR4/My- D88/NF-κB signal pathway to relieve inflammation.

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Publication Details

Journal
Current Drug Delivery
Published
2024-10-14
DOI
https://doi.org/10.2174/0115672018304338241003095955
Citations
3
Primary Topic
Oral health in cancer treatment
Type
article
Field-Weighted Citation Impact
1.26
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article

Fecal Microbiota Transplantation Induced by Wumei Pills Improves Chemotherapy-Induced Intestinal Mucositis in BALB/c Mice by Modulating the TLR4/MyD88/NF-κB Signaling Pathway

Lijiang Ji, Wu Hua, Feng Liu, Dongxue Lu et al.
3 citations
Current Drug Delivery
Oral health in cancer treatment
1.26
article

Fecal Microbiota Transplantation Induced by Wumei Pills Improves Chemotherapy-Induced Intestinal Mucositis in BALB/c Mice by Modulating the TLR4/MyD88/NF-κB Signaling Pathway

Lijiang Ji, Wu Hua, Feng Liu, Dongxue Lu, Jing Yan, Zhiguang Sun, Haixia Liu
article en
3 citations

Abstract

FMT can defend the intestinal mucosal barrier integrality by increasing the content of exercise fatty acids, and its mechanism may be in connection with its inhibition of TLR4/My- D88/NF-κB signal pathway to relieve inflammation.

Current Drug DeliveryVol. 22(7)
Nanjing University of Chinese Medicine (CN), Nanjing Traditional Chinese Medicine Hospital (CN), Jiangsu Provincial Academy of Traditional Chinese Medicine (CN), Changshu No.1 People's Hospital (CN), Nanjing University (CN)
Openalex Percentile: Top 18%
Oral health in cancer treatment
1.26
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Fecal Microbiota Transplantation Induced by Wumei Pills Improves Chemotherapy-Induced Intestinal Mucositis in BALB/c Mice by Modulating the TLR4/MyD88/NF-κB Signaling Pathway — Lijiang Ji, Wu Hua, et al. · Current Drug Delivery (2024) | TGRS Research Map | TGRS