Renin‐angiotensin blockade ameliorates the progression of glomerular injury in podocyte‐specific <i>Ctcf</i> knockout mice
These results suggest that RAS is involved in the development of glomerulopathy in podocyte-specific Ctcf knockout mice. Elucidation of the pathophysiology of podocyte-specific Ctcf knockout mice may provide new insights into the relationship between podocyte injury and chronic glomerulonephritis.
Authors
- Takashi Nagai (ORCID: https://orcid.org/0000-0001-8116-1751)
- Keisuke Fujioka (ORCID: https://orcid.org/0000-0001-9731-1515)
- Maki Urushihara (ORCID: https://orcid.org/0000-0002-6431-3239)
- Tomoki Hattori
- Teruyoshi Hirayama (ORCID: https://orcid.org/0000-0002-4407-9203)
- Niels Galjart (ORCID: https://orcid.org/0000-0003-1964-3607)
- Koji Yasutomo (ORCID: https://orcid.org/0000-0003-0411-2037)
- Shoji Kagami
- Hiroshi Kawachi
Institutions
- Erasmus MC (NL)
- Niigata University (JP)
- Tokushima University (JP)
Publication Details
- Journal
- Nephrology
- Published
- 2024-09-30
- DOI
- https://doi.org/10.1111/nep.14400
- Primary Topic
- Renal Diseases and Glomerulopathies
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- Japan Society for the Promotion of Science