Crosstalk between human endogenous retroviruses and exogenous viruses

Human endogenous retroviruses (HERVs) are remnants of ancient retroviral infections of human germ-line cells, which are mostly silenced during evolution, but could be de-repressed and play a pathological role. Infection with some exogenous viruses, including herpesviruses, HIV-1 and SARS-CoV-2, was demonstrated to induce the expression of HERV RNAs and proteins.

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Publication Details

Journal
Microbes and Infection
Published
2024-09-28
DOI
https://doi.org/10.1016/j.micinf.2024.105427
Citations
6
Primary Topic
Chromosomal and Genetic Variations
Type
article
Field-Weighted Citation Impact
3.60

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article

Crosstalk between human endogenous retroviruses and exogenous viruses

Claudia Matteucci, Antonella Minutolo, Branka Horvat, Gkikas Magiorkinis et al.
6 citations
Microbes and Infection
Chromosomal and Genetic Variations
3.60
article

Crosstalk between human endogenous retroviruses and exogenous viruses

Claudia Matteucci, Antonella Minutolo, Branka Horvat, Gkikas Magiorkinis, Edoardo Pizzioli, Emanuela Balestrieri
article en
6 citations

Abstract

Human endogenous retroviruses (HERVs) are remnants of ancient retroviral infections of human germ-line cells, which are mostly silenced during evolution, but could be de-repressed and play a pathological role. Infection with some exogenous viruses, including herpesviruses, HIV-1 and SARS-CoV-2, was demonstrated to induce the expression of HERV RNAs and proteins.

Microbes and InfectionVol. 27(5-6)
Université Claude Bernard Lyon 1 (FR), École Normale Supérieure de Lyon (FR), University of Rome Tor Vergata (IT), Centre National de la Recherche Scientifique (FR), Inserm (FR), National and Kapodistrian University of Athens (GR), Centre International de Recherche en Infectiologie (FR)
Horizon 2020 Framework Programme
Openalex Percentile: Top 8%
Chromosomal and Genetic Variations
3.60
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Crosstalk between human endogenous retroviruses and exogenous viruses — Claudia Matteucci, Antonella Minutolo, et al. · Microbes and Infection (2024) | TGRS Research Map | TGRS