Commentary on “Intra‐islet α‐cell Gs signaling promotes glucagon release”

The Gs receptor in pancreatic α cells promotes glucagon transcription and secretion via increasing cAMP. The Gi receptor in pancreatic α cells suppresses glucagon secretion via decreasing cAMP. The Gq receptor in pancreatic α cells promotes glucagon secretion via releasing Ca<sup>++</sup> from ER to cytoplasm.

Authors

Publication Details

Journal
Journal of Diabetes Investigation
Published
2024-09-26
DOI
https://doi.org/10.1111/jdi.14317
Primary Topic
Pancreatic function and diabetes
Type
article
Field-Weighted Citation Impact
0.00
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article

Commentary on “Intra‐islet α‐cell Gs signaling promotes glucagon release”

Tadahiro Kitamura
Journal of Diabetes Investigation
Pancreatic function and diabetes
article

Commentary on “Intra‐islet α‐cell Gs signaling promotes glucagon release”

Tadahiro Kitamura
article en

Abstract

The Gs receptor in pancreatic α cells promotes glucagon transcription and secretion via increasing cAMP. The Gi receptor in pancreatic α cells suppresses glucagon secretion via decreasing cAMP. The Gq receptor in pancreatic α cells promotes glucagon secretion via releasing Ca<sup>++</sup> from ER to cytoplasm.

Journal of Diabetes InvestigationVol. 15(12)
Zero hunger
Openalex Percentile: Top 69%
Pancreatic function and diabetes
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Commentary on “Intra‐islet α‐cell Gs signaling promotes glucagon release” — Tadahiro Kitamura · Journal of Diabetes Investigation (2024) | TGRS Research Map | TGRS