Transcriptomic Analysis of Cardiac Tissues in a Rodent Model of Coronary Microembolization
Our findings suggest that dysfunctions in mitochondrial activity and metabolism are important mechanisms for CME, and mitochondria-related DEGs may be potential therapeutic targets for CME.
Authors
- Yu Ding (ORCID: https://orcid.org/0000-0003-1246-2563)
- Haohao Lu (ORCID: https://orcid.org/0000-0002-8470-750X)
- Jinyu Huang (ORCID: https://orcid.org/0000-0003-4993-3197)
- Beibei Gao (ORCID: https://orcid.org/0000-0003-0288-0702)
- Zhaochang Jiang
Institutions
- Affiliated Hangzhou First People's Hospital, Westlake University, School of Medicine (CN)
- Second Affiliated Hospital of Zhejiang University (CN)
- Hangzhou Medical College (CN)
Publication Details
- Journal
- Journal of Inflammation Research
- Published
- 2024-09-01
- DOI
- https://doi.org/10.2147/jir.s469297
- Citations
- 1
- Primary Topic
- Cardiac Fibrosis and Remodeling
- Type
- article
- Field-Weighted Citation Impact
- 0.40
Funders
- National Natural Science Foundation of China
- Natural Science Foundation of Zhejiang Province