Granulocyte colony stimulating factor promotes scarless tissue regeneration

Mammals typically heal with fibrotic scars, and treatments to regenerate human skin and hair without a scar remain elusive. We discovered that mice lacking C-X-C motif chemokine receptor 2 (CXCR2 knockout [KO]) displayed robust and complete tissue regeneration across three different injury models: skin, hair follicle, and cartilage. Remarkably, wild-type mice receiving plasma from CXCR2 KO mice through parabiosis or injections healed wounds scarlessly. A comparison of circulating proteins using multiplex ELISA revealed a 24-fold higher plasma level of granulocyte colony stimulating factor (G-CSF) in CXCR2 KO blood. Local injections of G-CSF into wild-type (WT) mouse wound beds reduced scar formation and increased scarless tissue regeneration. G-CSF directly polarized macrophages into an anti-inflammatory phenotype, and both CXCR2 KO and G-CSF-treated mice recruited more anti-inflammatory macrophages into injured areas. Modulating macrophage activation states at early time points after injury promotes scarless tissue regeneration and may offer a therapeutic approach to improve healing of human skin wounds.

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Publication Details

Journal
Cell Reports
Published
2024-09-21
DOI
https://doi.org/10.1016/j.celrep.2024.114742
Citations
5
Primary Topic
Wound Healing and Treatments
Type
article
Field-Weighted Citation Impact
1.25

Funders

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article

Granulocyte colony stimulating factor promotes scarless tissue regeneration

Denis H. Y. Leung, Emmanuel Rapp, Emily Sheng, Thomas Leung et al.
5 citations
Cell Reports
Wound Healing and Treatments
1.25
article

Granulocyte colony stimulating factor promotes scarless tissue regeneration

Denis H. Y. Leung, Emmanuel Rapp, Emily Sheng, Thomas Leung, Stephen M. Prouty, Satish Sati, Parker Jones, Scott W. Korte, Anna E. Kersh, Jianhe Huang, Olivia Ahart, Casey A. Spencer, Christina Murphy
article en
5 citations

Abstract

Mammals typically heal with fibrotic scars, and treatments to regenerate human skin and hair without a scar remain elusive. We discovered that mice lacking C-X-C motif chemokine receptor 2 (CXCR2 knockout [KO]) displayed robust and complete tissue regeneration across three different injury models: skin, hair follicle, and cartilage. Remarkably, wild-type mice receiving plasma from CXCR2 KO mice through parabiosis or injections healed wounds scarlessly. A comparison of circulating proteins using multiplex ELISA revealed a 24-fold higher plasma level of granulocyte colony stimulating factor (G-CSF) in CXCR2 KO blood. Local injections of G-CSF into wild-type (WT) mouse wound beds reduced scar formation and increased scarless tissue regeneration. G-CSF directly polarized macrophages into an anti-inflammatory phenotype, and both CXCR2 KO and G-CSF-treated mice recruited more anti-inflammatory macrophages into injured areas. Modulating macrophage activation states at early time points after injury promotes scarless tissue regeneration and may offer a therapeutic approach to improve healing of human skin wounds.

Cell ReportsVol. 43(10)
Philadelphia VA Medical Center (US), University of Pennsylvania (US), Singapore Management University (SG)
U.S. Department of Veterans Affairs, Dermatology Foundation, University of Pennsylvania, Lisa Dean Moseley Foundation, National Institutes of Health, Penn Skin Biology and Diseases Resource-based Center, University of Pennsylvania
Zero hunger
Openalex Percentile: Top 18%
Wound Healing and Treatments
1.25
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