Craniotubular Dysplasia Ikegawa Type: Further Delineation of the Phenotype

Craniotubular Dysplasia Ikegawa type is a sclerosing bone disorder recently identified in five patients from four independent Indian families. It is caused by homozygous or compound heterozygous mutations in TMEM53. Deficient TMEM53 leads to overactive BMP signaling which promotes bone formation. Here, we present another three siblings with intronic mutations in TMEM53, identified by exome sequencing, from a Caucasian family. All three siblings displayed skeletal and radiographic features, similar to the earlier described individuals. All our patients had additional features such as cardiac and urogenital anomalies. Our results confirm the phenotype of CTDI. We discuss whether the additional features in our patients are separate from CTDI or reflect a broader spectrum of the syndrome.

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Publication Details

Journal
American Journal of Medical Genetics Part A
Published
2024-09-20
DOI
https://doi.org/10.1002/ajmg.a.63870
Citations
3
Primary Topic
Bone Metabolism and Diseases
Type
article
Field-Weighted Citation Impact
0.70
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Craniotubular Dysplasia Ikegawa Type: Further Delineation of the Phenotype

Babeth van Ommeren, Marie‐Lise C. van Veelen, Gijs van Haaften, Rúben Dammers et al.
3 citations
American Journal of Medical Genetics Part A
Bone Metabolism and Diseases
0.70
article

Craniotubular Dysplasia Ikegawa Type: Further Delineation of the Phenotype

Babeth van Ommeren, Marie‐Lise C. van Veelen, Gijs van Haaften, Rúben Dammers, Koen L.I. van Gassen, Jacques C. Giltay, Rolanda Baars, Irene M. J. Mathijssen, Richard van Jaarsveld, Maud Hoekstra
article en
3 citations

Abstract

Craniotubular Dysplasia Ikegawa type is a sclerosing bone disorder recently identified in five patients from four independent Indian families. It is caused by homozygous or compound heterozygous mutations in TMEM53. Deficient TMEM53 leads to overactive BMP signaling which promotes bone formation. Here, we present another three siblings with intronic mutations in TMEM53, identified by exome sequencing, from a Caucasian family. All three siblings displayed skeletal and radiographic features, similar to the earlier described individuals. All our patients had additional features such as cardiac and urogenital anomalies. Our results confirm the phenotype of CTDI. We discuss whether the additional features in our patients are separate from CTDI or reflect a broader spectrum of the syndrome.

American Journal of Medical Genetics Part AVol. 197(2)
Utrecht University (NL), Erasmus MC (NL), University Medical Center Utrecht (NL), Erasmus MC - Sophia Children’s Hospital (NL), Wilhelmina Children's Hospital (NL)
Good health and well-being
Openalex Percentile: Top 30%
Bone Metabolism and Diseases
0.70
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