The loss of dNK1/2 and EVT1 cells at the maternal–fetal interface is associated with recurrent miscarriage
The communication between EVT1 and dNK1/2 mediated the chemotaxis of EVT1 and facilitated regulation of endothelial cell death, initiating spiral artery remodeling. The loss of this specific cellular interaction may result in impaired decidualization, leading to recurrent miscarriage.
Authors
- Qiaoqiao Xu (ORCID: https://orcid.org/0000-0001-7315-5431)
- Yankai Xia (ORCID: https://orcid.org/0000-0003-0484-4035)
- Chuncheng Lu (ORCID: https://orcid.org/0000-0003-3689-9188)
- Wenjun Cheng
- Yun Fan (ORCID: https://orcid.org/0000-0002-0561-0292)
- Hong Qian (ORCID: https://orcid.org/0000-0001-7218-2681)
- Yuchen Zhang (ORCID: https://orcid.org/0000-0001-8944-5230)
- Yijun Yang (ORCID: https://orcid.org/0000-0002-9855-9616)
- Yingchun Gao
- Jiangnan Qiu
- Zhu Wu
- Can Shi (ORCID: https://orcid.org/0000-0003-1233-9039)
- Hui Wang (ORCID: https://orcid.org/0009-0006-3512-9308)
Institutions
- Ministry of Education of the People's Republic of China (CN)
- Xuzhou Medical College (CN)
- Nanjing Health and Health Commission (CN)
- Huaian First People’s Hospital (CN)
- Ministry of Education (KR)
- Second People’s Hospital of Huai’an (CN)
- Jiangsu Province Hospital (CN)
- Nanjing Medical University (CN)
Publication Details
- Journal
- Biology of Reproduction
- Published
- 2024-09-20
- DOI
- https://doi.org/10.1093/biolre/ioae136
- Citations
- 4
- Primary Topic
- Reproductive System and Pregnancy
- Type
- article
- Field-Weighted Citation Impact
- 0.96