STAT3 induces amygdala apoptosis by regulating the expression of stathmin in the rat model of posttraumatic stress disorder
The findings above suggest that SPS could potentially modulate the stathmin gene's expression by activating the STAT3 pathway, subsequently leading to apoptosis in amygdala cells. This sequence of events ultimately contributes to the PTSD rat model fear memory impairment.
Authors
- Shengxue Yu (ORCID: https://orcid.org/0000-0003-4064-089X)
- Wenqiang Liu (ORCID: https://orcid.org/0000-0002-2323-7962)
- Anqi Liu (ORCID: https://orcid.org/0000-0002-9788-2825)
- Shan Wei (ORCID: https://orcid.org/0000-0001-6211-7368)
- Yufei Wang (ORCID: https://orcid.org/0000-0002-9667-2105)
Institutions
- Perceptive Engineering (United Kingdom) (GB)
- Jinzhou Medical University (CN)
Publication Details
- Journal
- Folia Neuropathologica
- Published
- 2024-09-19
- DOI
- https://doi.org/10.5114/fn.2024.140830
- Primary Topic
- Stress Responses and Cortisol
- Type
- article
- Field-Weighted Citation Impact
- 0.00