COL12A1 Promotes Osteosarcoma Progression via the FAK/PI3K/AKT/mTOR Pathway

Our findings indicated that COL12A1 enhanced OS development by activating the FAK/PI3K/AKT/mTOR pathway, suggesting that COL12A1 could serve as a valuable biomarker for the prediction and identification of OS patients.

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Publication Details

Journal
Current Molecular Medicine
Published
2024-09-16
DOI
https://doi.org/10.2174/0115665240322280240903111159
Citations
5
Primary Topic
Cell Adhesion Molecules Research
Type
article
Field-Weighted Citation Impact
1.91
Controls
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article

COL12A1 Promotes Osteosarcoma Progression via the FAK/PI3K/AKT/mTOR Pathway

Yu Fu, Zhichao Gao, Lizhe Xu, Yun-Pu Zhang et al.
5 citations
Current Molecular Medicine
Cell Adhesion Molecules Research
1.91
article

COL12A1 Promotes Osteosarcoma Progression via the FAK/PI3K/AKT/mTOR Pathway

Yu Fu, Zhichao Gao, Lizhe Xu, Yun-Pu Zhang, Hai-xia Wang
article en
5 citations

Abstract

Our findings indicated that COL12A1 enhanced OS development by activating the FAK/PI3K/AKT/mTOR pathway, suggesting that COL12A1 could serve as a valuable biomarker for the prediction and identification of OS patients.

Current Molecular MedicineVol. 25(7)
Chifeng University (CN), Wuhan University (CN), Second Affiliated Hospital of Inner Mongolia Medical University (CN), Renmin Hospital of Wuhan University (CN)
Good health and well-being
Openalex Percentile: Top 13%
Cell Adhesion Molecules Research
1.91
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