Enhancer-promoter hubs organize transcriptional networks promoting oncogenesis and drug resistance

Recent advances in high-resolution mapping of spatial interactions among regulatory elements support the existence of complex topological assemblies of enhancers and promoters known as enhancer-promoter hubs or cliques. Yet, organization principles of these multi-interacting enhancer-promoter hubs and their potential role in regulating gene expression in cancer remain unclear. Here, we systematically identify enhancer-promoter hubs in breast cancer, lymphoma, and leukemia. We find that highly interacting enhancer-promoter hubs form at key oncogenes and lineage-associated transcription factors potentially promoting oncogenesis of these diverse cancer types. Genomic and optical mapping of interactions among enhancer and promoter elements further show that topological alterations in hubs coincide with transcriptional changes underlying acquired resistance to targeted therapy in T cell leukemia and B cell lymphoma. Together, our findings suggest that enhancer-promoter hubs are dynamic and heterogeneous topological assemblies with the potential to control gene expression circuits promoting oncogenesis and drug resistance.

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Publication Details

Journal
Nature Communications
Published
2024-09-14
DOI
https://doi.org/10.1038/s41467-024-52375-6
Citations
12
Primary Topic
Genomics and Chromatin Dynamics
Type
article
Field-Weighted Citation Impact
2.81

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article

Enhancer-promoter hubs organize transcriptional networks promoting oncogenesis and drug resistance

Robert B. Faryabi, Zora Modrušan, Yeqiao Zhou, Gregory W. Schwartz et al.
12 citations
Nature Communications
Genomics and Chromatin Dynamics
2.81
article

Enhancer-promoter hubs organize transcriptional networks promoting oncogenesis and drug resistance

Robert B. Faryabi, Zora Modrušan, Yeqiao Zhou, Gregory W. Schwartz, Noah Burget, Brent S. Perlman, Jelena Petrovic
article en
12 citations

Abstract

Recent advances in high-resolution mapping of spatial interactions among regulatory elements support the existence of complex topological assemblies of enhancers and promoters known as enhancer-promoter hubs or cliques. Yet, organization principles of these multi-interacting enhancer-promoter hubs and their potential role in regulating gene expression in cancer remain unclear. Here, we systematically identify enhancer-promoter hubs in breast cancer, lymphoma, and leukemia. We find that highly interacting enhancer-promoter hubs form at key oncogenes and lineage-associated transcription factors potentially promoting oncogenesis of these diverse cancer types. Genomic and optical mapping of interactions among enhancer and promoter elements further show that topological alterations in hubs coincide with transcriptional changes underlying acquired resistance to targeted therapy in T cell leukemia and B cell lymphoma. Together, our findings suggest that enhancer-promoter hubs are dynamic and heterogeneous topological assemblies with the potential to control gene expression circuits promoting oncogenesis and drug resistance.

Nature CommunicationsVol. 15(1)
University Health Network (CA), Cancer Research Institute (US), University of Toronto (CA), Princess Margaret Cancer Centre (CA), Penn Center for AIDS Research (US), UPMC Hillman Cancer Center (US), Genetic Technologies (Australia) (AU), University of Pennsylvania (US)
National Institutes of Health
Openalex Percentile: Top 9%
Genomics and Chromatin Dynamics
2.81
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