Long non-coding ribonucleic acid SNHG18 induced human granulosa cell apoptosis via disruption of glycolysis in ovarian aging
Altogether, our results revealed that folliculogenesis-related lncRNA SNHG18 participated in the pathogenesis of ovarian aging, which may provide novel biomarkers for ovarian function and new insights for the infertility treatment.
Authors
- Feiyan Zhao (ORCID: https://orcid.org/0000-0001-9103-5003)
- Jiaqi Wu (ORCID: https://orcid.org/0000-0001-7737-5597)
- Xuehan Zhao (ORCID: https://orcid.org/0009-0006-4129-1571)
- Ying Fang (ORCID: https://orcid.org/0000-0003-2313-9139)
- Xiaokui Yang (ORCID: https://orcid.org/0000-0001-8592-9262)
- Yan Long (ORCID: https://orcid.org/0000-0001-9274-7950)
- Zhimin Xin (ORCID: https://orcid.org/0000-0001-9051-2242)
- Cong Wang (ORCID: https://orcid.org/0000-0002-4100-4222)
Institutions
- Capital Medical University (CN)
- Chinese Academy of Sciences (CN)
- Beijing Chao-Yang Hospital, Capital Medical University (CN)
- Beijing Obstetrics and Gynecology Hospital (CN)
- University of Chinese Academy of Sciences (CN)
- Institute of Zoology (CN)
Publication Details
- Journal
- Journal of Ovarian Research
- Published
- 2024-09-13
- DOI
- https://doi.org/10.1186/s13048-024-01510-4
- Citations
- 3
- Primary Topic
- Cancer-related molecular mechanisms research
- Type
- article
- Field-Weighted Citation Impact
- 0.86
Funders
- National Natural Science Foundation of China
- National Key Research and Development Program of China