Case Report: Glossopharyngeal Allodynia-Related Odynophagia and Dysphagia Post Anterior Cervical Discectomy and Fusion Managed with Glossopharyngeal Nerve Block
Dysphagia after anterior cervical spine surgery has a 5% to 15% incidence beyond 1-year postsurgery, often attributed to mechanical factors such as pharyngeal thickening and epiglottis inversion. Despite normal neurological examination and electromyography, nerve distortion related to stretching also remains a possibility in these patients and may cause allodynia resulting in odynophagia and dysphagia. Current treatment options for dysphagia after anterior cervical discectomy and fusion are limited to local intraoperative steroid injections and tracheal traction exercises. In our patient, a glossopharyngeal nerve block was effectively used to manage the glossopharyngeal allodynia, thereby reducing the odynophagia and dysphagia, ultimately enhancing oral tolerance.
Authors
- Michael E Schatman (ORCID: https://orcid.org/0000-0002-1019-0413)
- Reda Tolba
- Alan D. Kaye (ORCID: https://orcid.org/0000-0003-2464-0187)
- Christopher Louis Robinson (ORCID: https://orcid.org/0000-0002-6276-9056)
- Alexandra Fonseca (ORCID: https://orcid.org/0000-0001-9735-3184)
- Vijay Raj (ORCID: https://orcid.org/0000-0001-6937-9053)
- Linda Kollenburg (ORCID: https://orcid.org/0009-0004-1044-2838)
- Krishna Pokuri
- Fahed Alrowaily
- Peter van der Meer
Institutions
- Brigham and Women's Hospital (US)
- Tufts Medical Center (US)
- Beth Israel Deaconess Medical Center (US)
- Cleveland Clinic (US)
- Harvard University (US)
- Radboud University Nijmegen (NL)
- Southern New Hampshire Medical Center (US)
- King Saud Medical City (SA)
- Radboud University Medical Center (NL)
- New York University (US)
- Louisiana State University Health Sciences Center Shreveport (US)
Publication Details
- Journal
- A&A Practice
- Published
- 2024-09-01
- DOI
- https://doi.org/10.1213/xaa.0000000000001849
- Primary Topic
- Cervical and Thoracic Myelopathy
- Type
- article
- Field-Weighted Citation Impact
- 0.00