Vaccine induced mucosal and systemic memory NK/ILCs elicit decreased risk of SIV/SHIV acquisition

SIV and HIV-based envelope V1-deleted (ΔV1) vaccines, delivered systemically by the DNA/ALVAC/gp120 platform, decrease the risk of mucosal SIV or SHIV acquisition more effectively than V1-replete vaccines. Here we investigated the induction of mucosal and systemic memory-like NK cells as well as antigen-reactive ILC response by DNA/ALVAC/gp120-based vaccination and their role against SIV/SHIV infection. ΔV1 HIV vaccination elicited a higher level of mucosal TNF-α<sup>+</sup> and CD107<sup>+</sup> memory-like NK cells than V1-replete vaccination, suggesting immunogen dependence. Mucosal memory-like NK cells, systemic granzyme B<sup>+</sup> memory NK cells, and vaccine-induced mucosal envelope antigen-reactive IL-17<sup>+</sup> NKp44<sup>+</sup> ILCs, IL-17<sup>+</sup> ILC3s, and IL-13<sup>+</sup> ILC2 subsets were linked to a lower risk of virus acquisition. Additionally, mucosal memory-like NK cells and mucosal env-reactive IFN-γ<sup>+</sup> ILC1s and env- reactive IL-13<sup>+</sup> ILC2 subsets correlated with viral load control. We further observed a positive correlation between post-vaccination systemic and mucosal memory-like NK cells, suggesting vaccination enhances the presence of these cells in both compartments. Mucosal and systemic memory-like NK cells positively correlated with V2-specific ADCC responses, a reproducible correlate of reduced risk of SIV/HIV infection. In contrast, an increased risk was associated with the level of mucosal PMA/Ionomycin-induced IFN-γ<sup>+</sup> and CD107<sup>+</sup> NKG2A<sup>-</sup>NKp44<sup>-</sup> ILCs. Plasma proteomic analyses demonstrated that suppression of mucosal memory-like NK cells was linked to the level of CCL-19, LT-α, TNFSF-12, and IL-15, suppression of systemic env-reactive granzyme B<sup>+</sup> memory-like NK cells was associated with the level of OLR1, CCL-3, and OSM, and suppression of IL-17<sup>+</sup> ILCs immunity was correlated with the level of IL-6 and CXCL-9. In contrast, FLT3 ligand was associated with promotion of protective mucosal env-reactive IL-17<sup>+</sup> responses. These findings emphasize the importance of mucosal memory-like NK cell and envelope- reactive ILC responses for protection against mucosal SIV/SHIV acquisition.

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Publication Details

Journal
Frontiers in Immunology
Published
2024-09-05
DOI
https://doi.org/10.3389/fimmu.2024.1441793
Citations
8
Primary Topic
Immune Cell Function and Interaction
Type
article
Field-Weighted Citation Impact
1.92

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article

Vaccine induced mucosal and systemic memory NK/ILCs elicit decreased risk of SIV/SHIV acquisition

Luca Schifanella, Isabela Silva de Castro, Genoveffa Franchini, Massimiliano Bissa et al.
8 citations
Frontiers in Immunology
Immune Cell Function and Interaction
1.92
article

Vaccine induced mucosal and systemic memory NK/ILCs elicit decreased risk of SIV/SHIV acquisition

Luca Schifanella, Isabela Silva de Castro, Genoveffa Franchini, Massimiliano Bissa, Mohammad Arif Rahman
article en
8 citations

Abstract

SIV and HIV-based envelope V1-deleted (ΔV1) vaccines, delivered systemically by the DNA/ALVAC/gp120 platform, decrease the risk of mucosal SIV or SHIV acquisition more effectively than V1-replete vaccines. Here we investigated the induction of mucosal and systemic memory-like NK cells as well as antigen-reactive ILC response by DNA/ALVAC/gp120-based vaccination and their role against SIV/SHIV infection. ΔV1 HIV vaccination elicited a higher level of mucosal TNF-α<sup>+</sup> and CD107<sup>+</sup> memory-like NK cells than V1-replete vaccination, suggesting immunogen dependence. Mucosal memory-like NK cells, systemic granzyme B<sup>+</sup> memory NK cells, and vaccine-induced mucosal envelope antigen-reactive IL-17<sup>+</sup> NKp44<sup>+</sup> ILCs, IL-17<sup>+</sup> ILC3s, and IL-13<sup>+</sup> ILC2 subsets were linked to a lower risk of virus acquisition. Additionally, mucosal memory-like NK cells and mucosal env-reactive IFN-γ<sup>+</sup> ILC1s and env- reactive IL-13<sup>+</sup> ILC2 subsets correlated with viral load control. We further observed a positive correlation between post-vaccination systemic and mucosal memory-like NK cells, suggesting vaccination enhances the presence of these cells in both compartments. Mucosal and systemic memory-like NK cells positively correlated with V2-specific ADCC responses, a reproducible correlate of reduced risk of SIV/HIV infection. In contrast, an increased risk was associated with the level of mucosal PMA/Ionomycin-induced IFN-γ<sup>+</sup> and CD107<sup>+</sup> NKG2A<sup>-</sup>NKp44<sup>-</sup> ILCs. Plasma proteomic analyses demonstrated that suppression of mucosal memory-like NK cells was linked to the level of CCL-19, LT-α, TNFSF-12, and IL-15, suppression of systemic env-reactive granzyme B<sup>+</sup> memory-like NK cells was associated with the level of OLR1, CCL-3, and OSM, and suppression of IL-17<sup>+</sup> ILCs immunity was correlated with the level of IL-6 and CXCL-9. In contrast, FLT3 ligand was associated with promotion of protective mucosal env-reactive IL-17<sup>+</sup> responses. These findings emphasize the importance of mucosal memory-like NK cell and envelope- reactive ILC responses for protection against mucosal SIV/SHIV acquisition.

Frontiers in ImmunologyVol. 15
National Cancer Institute (US)
U.S. Department of Health and Human Services, National Institutes of Health, National Cancer Institute, National Institute of Allergy and Infectious Diseases
Openalex Percentile: Top 14%
Immune Cell Function and Interaction
1.92
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